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We are far detached from the protocols of the early script and for good cause

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It is noteworthy that in the present study we used OA, a selective agonist of the TGR5 receptor, rather than native bile salts, to examine the regulation of PYY and GLP-1 release (Sato et al., 50 for TGR5 ligand stimulated cAMP formation and PI hydrolysis is close to the luminal bile acids concentrations and suggests that under physiological conditions it is likely that L cells would be activated by postprandial bile acids and induce GLP-1 and PYY release