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In Alzheimer's disease (AD), the disruption of MAMs enhances inflammatory signaling and impairs the cellular response to ER stress ( 5.4 Mitochondrial dysfunction as an upstream driver of amyloid and tau pathology A significant shift in Alzheimer's disease (AD) research is represented by the Mitochondrial Cascade Hypothesis, which posits that mitochondrial dysfunction is the primary driver of AD pathophysiology, rather than a secondary consequence of Tau or A accumulation ( Importantly, this theory posits that the production of A is influenced by mitochondrial dysfunction triggered by toxins
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