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VRK2 expression stratifies the response of PC to GSH-targeted treatment To further investigate the differential sensitivity to GSH inhibitors between VRK2-expressing and VRK2-deficient pancreatic tumors in vivo, we developed orthotopic pancreatic cancer models using Pan02 cells (Fig

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Despite its advantages, GST is a dimeric enzyme, and its crystal structure reveals four exposed cysteine residues that can be easily oxidized to form aggregates 20

Compared to M-CuP, D-CuP exhibited significantly higher proliferative activity at lower concentrations, which was more conducive to wound healing