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If MCF-7:2A cells do indeed possess high levels of GSH, then it is possible that the use of BSO as a single agent might be able to sensitize these cells to estrogen-induced apoptosis

Date from in vitro study showed significantly increased the GSH levels and GSH/GSSG ratio in normal human epidermal keratinocytes with presence of GAP, the protective effects of GAP against oxidative stress were superior to those of ascorbic acid

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