None of them addresses the actual claim being sold to biohackers, health conscious individuals or athletes which is that BPC-157 enhances recovery, repair, or longevity in healthy people
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concurrently, BNIP3 recruits Parkin to mitochondria to initiate autophagic flux, whereas NIXas a ubiquitination substrate of Parkinundergoes conformational remodeling post-modification to specifically engage adaptor protein NBR1, ultimately enabling targeted autophagic degradation of compromised mitochondria (Wang et al., 2023)
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