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Moreover, radon exposure has been linked to disrupted redox balance and impaired nitric oxide (NO) signaling, both of which are pivotal in maintaining vascular tone and endothelial function

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This inhibition preserves cellular NAD+ levels by reducing their consumption, simultaneously increasing GLUT4 transporter expression and activating sirtuin 1, known as the "longevity gene." The compound exhibits high membrane permeability and selectivity without affecting related methyltransferases or NAD+ salvage pathway enzymes, creating targeted metabolic effects while maintaining cellular safety