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Given their multifaceted involvement in CNS pathology, a reactive form of disease-associated astrocytes has emerged as promising, but technically challenging, targets for therapeutic intervention (Liddelow et al., 2017

As with other cell types, high glucose-driven impairment in astrocytes is mediated by overproduction of reactive oxygen species and activation of nuclear factor-B and signal transducer and activator of transcription 3 inflammatory pathways

This precision approach transforms the decision to switch from a reactive response to inadequate results into a proactive, data-driven strategy