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Although the precise pathophysiology of PEM in PCC remains unclear, previous research on ME/CFS, along with current pathological evidence from PCC, suggests that it is not the result of a single system dysfunction, but rather involves widespread pathophysiological alterations

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If thats true, then ME/CFS is not alone low levels of glutathione have been implicated in many central nervous system diseases (multiple sclerosis, autism, Alzheimers disease, Parkinsons disease, schizophrenia, and bipolar disease) as well

Although the reasons for this gender-specific outcome remain unknown, our findings underscore the nuanced and complex interplay of glutathione regulation in the context of lymphomagenesis and its potential therapeutic implications