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These findings suggest that GLP-1 receptor agonists may have kidney-protective effects in lupus nephritis, likely due to their anti-inflammatory properties

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This could be due to improved perfusion, or it could be a direct inotropic effect of GLP-1

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Results of a study indicated that chemotherapeutic agents-induced oxidative stress caused TRPA1 activation instead of direct channel targeting (Nassini et al., 1

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Maffulli et al (2018) also found similar improvements following a course of shockwave treatment

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