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The studies began with the determination of the optimal dosing for Cagrilintide in both chow-fed and high-fat diet (HFD)-fed wild-type (WT) mice, followed by analyses of its receptor dependence using receptor activity-modifying protein (RAMP) knockout (KO) models, and mechanistic studies involving neuronal activation and gene expression profiling [2]

10.1186/s42358-022-00252-5 96 MaJ.-D.JingJ.WangJ.-W.YanT.LiQ.-H.MoY.-Q.et al (2019)

None of the tumor volumes of mice in the current study transgressed these regulations

decreased islet beta-cell proliferation and insulin secretion, and insulin receptor knockout in neurons increase appetite