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Nevertheless, understanding the potential mechanisms that might explain these patient-reported experiences and distinguishing them from more serious circulatory concerns is essential for both patients and healthcare professionals

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We think that, by modulating cerebrospinal fluid pressure and reducing intracranial venous sinuses compression, these drugs produce a decrease in the release of calcitonin gene-related peptide (CGRP), a key migraine-promoting peptide, Dr Braca explained