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Firstly, activated NF-B and NLRP3 inflammasome in DN promotes inflammation and increases fibronectin and collagen deposition in kidney, 41,42 leading to thickening glomerular basement membrane, glomerular sclerosis, podocyte damage, and ultimately kidney fibrosis

In multiple sclerosis models, GLP-1 receptor activation reduces pathogenic Th1 and Th17 cell activity, inhibits microglial activation (the brain equivalent of macrophage inflammation), and may enhance remyelination processes

This article explores the mechanisms behind post-injection discomfort, common side effects, and practical management strategies aligned with UK clinical guidance