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It has been reported that AP can alleviate the degree of liver injury induced by D-galactosamine (D-GalN)/lipopolysaccharide (LPS) by increasing the levels of hepatic Nrf-2 and PPAR protein expressions as well as superoxide dismutase, catalase, glutathione s-transferase and glutathione reductase activities and decreasing the levels of hepatic NF-B protein expression and TNF- (Zhou R

ApoE / mice treated with liraglutide 400 g/day for 4 weeks exhibited increased AMP-activated protein kinase (AMPK) phosphorylation in the aortic wall, as measured by immunohistochemistry

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