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Mechanistically, mitochondrial dysfunction, as discussed above, is an increasingly recognized contributor, particularly in diabetic nephropathy, in which mitochondrial ROS production exceeds the local antioxidant capacity [13,34]

Therefore, oxidative cell death mechanisms offer the potential to selectively target and eliminate cancer cells while sparing healthy tissues, thereby minimizing the adverse effects commonly associated with traditional therapies

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Effects on Fat Mass The majority of studies examining GLP-1 RAs have shown a significant reduction in total body fat mass