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These findings suggest that glutamate/GABA imbalance, particularly within the corticostriatal pathway, may serve as a key pathophysiological mechanism and potential biomarker in SHANK3-associated ASD

In addition, MTHFD2 knockdown cells suffered from ferroptosis and exhibited remarkable up-regulation of intracellular ROS, lipid peroxidation, and MDA levels, which led to tumor cell impairment