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These appeared as having modulatory effects on NO-system (NO-release, NOS-inhibition, NO-over-stimulation all affected), controlling vasomotor tone and the activation of the Src-Caveolin-1-eNOS pathway and modulatory effects on the prostaglandins system (BPC 157 counteracted NSAIDs toxicity, counteracted bleeding, thrombocytopenia, and in particular, leaky gut syndrome)

Among these, eosinophils stand out as important yet underappreciated players in IBD, often overshadowed by the emphasis on neutrophils and lymphocytes in intestinal inflammation (1)

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