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GSH appears to be involved in the crosstalk between ferroptosis and autophagy

[64], which involved a co-culture of primary astrocytes with the bEnd.3 endothelial cell line, incubation with exendin-4 reduced the levels of the astrocyte-derived vascular endothelial growth factor (VEGF-A), matrix metalloproteinase-9 (MMP-9), chemokine monocyte chemoattractant protein-1 (MCP-1), and the chemokine C-X-C motif ligand 1 (CXCL-1), induced by oxygenglucose deprivation (OGD), and subsequently led to the increased expression of tight junction proteins in the bEnd.3 cells

Brownsey RW, Boone AN, Elliott JE, Kulpa JE, Lee WM

Accessed 12/17/2024, Schulz R, Schluter KD, Laufs U