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Description

Glucose-dependent insulinotropic polypeptide (GIP) inhibits bone resorption independently of insulin and glycemia

1 Introduction Ferroptosis was originally defined as iron-mediated lipid peroxidation damage, which is closely related to the physiological and pathological mechanisms of the ovary

In particular, activation of the GPCR, bile acid (GPBA) receptor, increases Lcell differentiation and elicits substantial GLP1 secretion (Lund et al., 2020)