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Dysregulation of these pathways contributes to impaired insulin secretion, altered lipid homeostasis, hyperglycemia, and metabolic dysfunction observed in obesity and type 2 diabetes [1]

Subsequent animal studies confirmed dose-dependent GH pulse amplification, additive effects with GHRH, and a favorable tolerability profile with repeat administration without tachyphylaxis (receptor desensitization) at doses studied

ABCG2 and ABCC4 utilize ATP hydrolysis to expel uric acid from the cell, and the urate efflux mediated by both depends on the intracellular urate concentration [80, 81]

10.4103/1673-5374.265562 68 SweeneyM