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In conclusion, we show that APAP toxicity in HepG2 cells is largely independent of oxidative stress, and is linked instead to a decoupling of glycolysis from the TCA cycle, lactic acidosis, reduced NADPH production, and subsequent suppression of the anabolic pathways required for rapid growth

Although the difference between the groups was not statistically significant, it suggests a slightly greater improvement in grip strength in the trans-osseous suture group