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So we looked beneath the surface of all-cause pancreatitis, at pancreatitis by specific cause.When we separated pancreatitis by cause, two opposing signals emerged: Increased risk of drug-induced pancreatitis: Reduced risks of alcohol-induced pancreatitis and triglyceride-induced pancreatitis These opposing forces cancelled each other out.This is why prior studies that lumped all pancreatitis together kept coming back null

Notably, PPAR alone is not sufficient to restore redox homeostasis and promote the M2 phenotype, as demonstrated by the exacerbation of neuroinflammatory cytokine production in NRF2-deficient mice challenged with LPS (Innamorato et al., 2008)

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