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The gpoA, tpxD, ahpD , and gshT mutants exhibited increased susceptibility to H 2 O 2 , and also impaired intracellular survival in pneumocytes, macrophages, and neutrophils compared to the wild-type strain

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Preclinical support for the potential of these drugs to alleviate IR damage has been shown for SOD@ARA290-HBc in RILI and for pMnSOD in radiation-damaged keratinocytes, demonstrating the translational potential for both of these antioxidant therapeutics [69, 77]