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By investigating specific rate-limiting enzymes and sulfur pathway variations, we can begin to create pharmacological agents to treat associated diseases

Strategies such as combining copper ionophores with ferroptosis inducers, or superimposing DSF/Cu regimens on a background of GSH depletion or p53 activation, could overcome resistance to single-death modalities and deliver a potent ferroptosis-cuproptosis double punch to OSCC

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(2009) examined the downstream effects of CJC-1295 DAC-induced GH/IGF-1 axis activation on the serum proteome, identifying changes in proteins related to lipid metabolism, complement activation, and coagulation pathways