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Increased binding was present in the hippocampus, amygdala, thalamus, midbrain, pons, and cingulate cortex, although the exact area of the cingulate cortex was not defined

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A subset of dedifferentiated -cells were at early stage to convert into -cell in LOsG-treated animals As a subset of FoxO1-deficient -cells adapts the -cell fate, resulting in hyperglucagonemia 22 , we determined pancreatic glucagon mRNA in LOsG-treated -cells, which were associated with low or absent FoxO1 expression