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Glutathione transferase activities in renal carcinomas and adjacent normal renal tissues: factors influencing renal carcinogenesis induced by xenobiotics

Rather than functioning as isolated antioxidant modules, these pathways form a highly interconnected redox buffering network in which compensatory capacity, metabolic flux, and iron availability collectively determine ferroptotic susceptibility

This phenomenon is characterized by an enhanced uptake of glucose and its subsequent conversion to lactic acid, even when oxygen is readily available [10]

An RT negative control (sample with RNA but without reverse transcriptase) was used during RT