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For example, research on B12-deficient individuals indicates significant improvements in memory and attention span with regular supplementation

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Guitton N, Brouazin-Jousseaume V, Dupaix A, Jgou B, Chenal C

Mutations in critical transporter and enzyme genes disrupt these steps and underlie a spectrum of neurological disorders collectively termed riboflavin-responsive neurometabolic diseases. A representative example is riboflavin transporter deficiency (RTD), caused by loss-of-function mutations in SLC52A2 and SLC52A3 , which encode the riboflavin transporters RFVT2 and RFVT3, respectively