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While it is primarily known for its tissue repair capabilities , its relevance to autoimmune disease extends into two specific areas: remyelination in multiple sclerosis and general tissue repair in autoimmune-damaged organs

The presence of this mechanism is supported by data from Cui et al., who found elevated hepcidin and ferritin levels, but a reduced hepcidin/ferritin ratio compared to healthy controls in a study including 107 MDS patients without prior transfusions ( ex vivo

they may act as starting events in PD pathogenesis [22]

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