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Activation of the PI3K/AKT pathway enhances neurogenesis, LTP, and mitochondrial function with a decrease in apoptosis and microglial activation

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259 Furthermore, endotoxin dose-dependent switch of NAD + biosynthesis pathways from NAMPT-dependent salvage to IDO1-dependent de novo biosynthesis maintains the nuclear NAD + pool, which promotes SIRT1-directed epigenetic regulation of immune tolerance