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The oxidative stress that occurs in the melanocyte, as mentioned before, is possibly the autoimmunity trigger in vitiligo.63 The communication between the melanocyte and the innate immune system seems to occur through the secretion of exosomes, which contain melanocyte-specific antigens, miRNAs, heat shock proteins, and DAMPs (damage-associated molecular patterns).64 These exosomes deliver autoantigens to dendritic cells, which undergo maturation into antigen-presenting cells.70 The DAMP with the highest evidence of association with vitiligo to date is HSP70 (heat shock protein 70).66 HSP70 is a protein from the intracellular chaperone family whose function is to prevent the incorrect folding of other proteins and their aggregation

The adult mammalian heart is notorious for its incapacity to regenerate after ischemia-reperfusion or myocardial infarction injuries, ultimately leading to the development of heart failure

27 The other RCT found no significant mean differences between B12 and placebo

Dr Kohli is currently practicing as Consultant Internal Medicine at Sitaram Bhartia Institute of Science and Research and Apollo Clinic in East of Kailash