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Following this protocol, the developed redox profiling method demonstrated high precision with intra-assay coefficients of variation (CV%) for each redox metabolite measured from frozen blood samples (Supplementary Tables 2 and 3)
Rosasco SL, Beard JK, Hallford DM, Summers AF
Subsequent research indicated that genistein reduces oxidative stress and myogenic apoptosis through various mechanisms, including the regulation of ROS, lipid peroxidation, Bcl-2, and the apoptosis marker caspase-3 via the PI3K-Akt and ERK1/2 MAPK signaling pathways, with its effects remaining unaffected by estrogen receptor antagonists [28]