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B 112 , 1651716524 (2008)

Notably, anticardiolipin (antiCL) autoantibodies trigger the formation of NETs via Fc receptor IIa (FcRIIa)dependent mechanisms, consequently facilitating a thromboticinflammatory cascade in antiphospholipid syndrome (APS) [171]

In addition, these chemokines secreted by macrophages enhance the recruitment of various immune cells, in which infiltrated monocytes can differentiate into different macrophage subtypes and play an important role in renal CaOx crystal formation [3]