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Endotoxin contamination: Bacterial byproducts that can cause fever and inflammatory reactions

Are there other mechanisms of cell death mediated by lysosomal damage, in addition to the release of cathepsins and hydrolases

In response to oxidative stress, neurons undergo overactivation of the enzyme poly (ADP-ribose) polymerase-1 (PARP1) to prevent DNA damage, which decreases intracellular NAD and ATP levels, compromising the efficiency of glycolysis and oxidative phosphorylation, affecting the ability of neurons to generate ATP (200, 203)

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