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GI Amyloidosis: Pathophysiology and Clinical Features The gastrointestinal symptoms of amyloidosis arise from the accumulation of amyloid protein fibrils in the GI tract, disrupting normal bowel function [6]

Despite the moderate sample size, the pooling strategy effectively reduced within-group variation, revealing statistically significant differences in multiple key quality parameters such as sperm motility and antioxidant enzyme activities [46]

Correspondingly, they found that plaque pathology progressed with age, from no plaques at 2 months to sparse plaques in the subiculum at 10 months, and abundant plaques in hippocampus subiculum at 20 months old 3xTd-AD mice (Lambracht-Washington et al., 2023)

People may have one or two mutations, or no mutations, of the MTHFR gene