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This interaction leads to the dephosphorylation of IB kinase, preventing the activation of NF-B, a transcription factor that regulates genes involved in immune responses, inflammation, and cell survival ( Through these combined mechanisms, ICP34.5 serves as a critical factor in HSV-1 pathogenesis by supporting viral replication, evading multiple immune pathways, and altering host cellular functions

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"L-Carnitine." Data on file

The effects of canagliflozin in type 2 diabetes in subgroups defined by population-specific body mass index: insights from the CANVAS program and CREDENCE trial