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Regarding intracellular roGFP2-based structure-function analysis of glutaredoxins, we now showed that the GSSG-dependent oxidation of the fused glutaredoxin moiety (and not the glutathionylation of the roGFP2 moiety) is rate-limiting unless saturating GSSG concentrations are present

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Point estimates in that study were of similar magnitude for duration of drug use 3 years.7 Given that a potential effect of GLP1 receptor agonists on thyroid cancer is unlikely to emerge after short term use, these findings might indicate the presence of confounding

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