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diabetes 2019;68

Accordingly, Masson staining revealed that recombinant CXCL10 treatment sharply increased fibrosis in the CXCL10 group, whereas IL18R1 antibody treatment reversed fibrosis to levels close to those of the control in the 10+18ab group (Fig

Similar to the systemic calcium signal transmission upon GSH treatment, Glu-induced [Ca 2+ ] cyt propagation in systemic leaves were strongly inhibited in GCaMP3/glr3.3 while remained unaffected in GCaMP3/glr3.6 (Supplementary Fig