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After the first clinical report of APAP toxicity [22], investigators at the US National Institute of Health developed a mouse model of APAP-induced hepatotoxicity, which mimicked the serious liver injury observed in humans [79]

Archived from the original on April 2, 2015

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It has been suggested that there are different mechanisms that depend on variables, such as Hcy concentration or duration of the HHcy state, according to which Hcy affects the Keap1Nrf2 pathway, thus leading to activation, or inhibition, of enzymes involved in protection against ROS