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75 Conversely, forced overexpression of CDO1 in breast cancer cells shifted the flux from glutathione synthesis towards cysteine catabolism, consequently increasing ROS levels and leading to reduced cell viability and growth

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Besides, in rats with inflamed intestines, PER downregulated the toll-like receptor 4 (TLR4), NF-B, and c-Fos/c-Jun pathways at the molecular level (Sayed et al

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