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Additionally, some peptides exhibit DPP-IV inhibitory effects, prolonging the half-life of incretin hormones, thus supporting glycemic control and offering therapeutic potential in type 2 diabetes mellitus (T2DM) [75, 76]

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Therefore, in the environment where MSU is present, promoting phagocytosis of MSU or MSU protein complexes by macrophages, thereby accelerating the metabolism of MSU from the site of injury is a potentially effective way to inhibit inflammation generation

We conclude that, while solutions based on homosynaptic plasticity may help balance synaptic changes (see Chistiakova et al., 2014, 2015