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These nonantimicrobial sulfonamides do not undergo metabolism to the N 4 -hydroxylated metabolite associated with SJS and will not bind to IgE at the N 1 position and, therefore, are unlikely to cause cross-reactivity, even in patients who have experienced type 1 hypersensitivity or serious non-type-1 hypersensitivity reactions to sulfonamide antimicrobial agents

doi: 10.1053/j.gastro.2011.04.052

Tiglyl-CoA is metabolized to 2-methyl-3-hydroxybutyryl-CoA (also identified as -methyl--hydroxybutyryl-CoA) by the enzyme often identified as crotonase

Peripheral blood neutrophils from COPD patients are also activated and release ROS, particularly during acute exacerbations [23]