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Since -catenin is not degraded by UPS, it may enter the nucleus and help the fibrogenic genes' transcription, which leads to the development of renal fibrosis[3]

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Inflammatory eosinophils II showed loss of early activation markers and further intensified the expression of inflammatory genes, while inflammatory eosinophils III downregulated inflammatory genes and increased mitochondrial gene expression ( mt-Co3 , mt-Nd1 ), suggesting metabolic adaptation

Intriguingly, in the animal nervous system, GSH has been identified as a neuromodulator/neurotransmitter 34,35,36 , suggesting a potential conceptual parallel in GSH-mediated signal transmission across different kingdoms of life