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This study emphasizes the age- and cell type-related epigenomic changes, including an excessive modification of H3K4me3 at developmental genes in the prefrontal cortex (PFC) neurons of newborns, which gradually diminishes after birth ( As highlighted throughout this discussion, the pathogenesis of numerous neurodevelopmental disorders is intricately tied to disruptions in histone methylation patterns, with many of these changes arising from loss-of-function mutations in key regulatory factors
