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Endometriosis is associated with increased activated pelvic macrophages, T-lymphocytes, and natural killer (NK) cells in the peritoneal environment, driving an inflammatory cascade ( The endometrial environment is further compromised by hormonal asynchrony between the endometrium and embryo, resulting from fluctuations in serum progesterone and estradiol levels

its activity appears permissive of healing processes already underway

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This could facilitate a deeper understanding of the fundamentals of the metabolic-immune crosstalk-regulated pathogenesis of MS