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Since IL-17 receptor A (IL-17RA) has been shown to mediate communication from the meninges to the cortex 47 , we next examined whether the beneficial effect of peripherally targeting CD38 could be mediated in part through modulation of cortical IL-17RA and IL-1 signaling, which we observed to drive T H 17 pathogenicity

A further longitudinal transcriptome analysis, which emphasized robust T cell immunity during recovery from COVID-19 showed that, during recovery, there was a significant downregulation of humoral immunity and type I interferon response, alongside upregulation in genes involved in T cell activation and differentiation