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This study generated a metabolite model that could predict clinical outcomes, quantified by Therapy Intensity Level (TIL), in the initial and validation cohorts based on the levels of the ketogenic AA isoleucine, ketone bodies (-hydroxybutyrate and acetoacetate), medium chain fatty acids (dodecanoic acid, suberic acid, 2-hydroxyoctanoic acid), medium chain fatty acid derivatives (10-hydroxydecanoic acid, 8-hydroxyoctanoic acid), and butyrylcarnitine (Table 2) [46]

These enhanced effects are attributed to the action of dual receptor activation, though the precise mechanisms underlying GIP and GLP-1 receptor crosstalk remain an active area of research

In addition, Alzheimers takes decades to develop but the observational studies suggested that the GLP-1 drugs had an effect too fast for the drugs to be the real cause