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The insulin-sensitizing effects of GLP-1/GIP agonism versus the insulin-desensitizing effects of GH elevation, modulated by glucagon-driven hepatic glucose output

Preclinical data also indicates a promising future in neuroprotection and most likely to expand its clinical utility into neurodegenerative pathologies

For instance, METTL14-mediated m^6A methylation accelerates GPX4 mRNA degradation, thereby enhancing ferroptosis susceptibility in IL-1-stimulated chondrocytes and OA cartilage