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In fact, many genes coding for sulfate transporters and for enzymes of the primary assimilation pathway are induced at the transcriptional level by sulfur deficiency whereas reduced sulfur-containing compounds, such as hydrogen sulfide (H 2 S), cysteine, S-adenosyl-methionine (SAM) and glutathione, exert inhibitory effects at the post-translational level (Kopriva and Koprivova, 2004

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Combining ferroptosis-inducing inducers with bio-nanotechnology for tumor therapy has broad application prospects [212], but there are still many problems to be solved in clinical ferroptosis-based nanotherapy

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