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Studies show that NNMT inhibition by 5-amino-1-methylquinolinium significantly reduces nicotinamide metabolism, increases NAM accumulation, improves NAD+ synthesis efficiency by over 30%, and markedly increases mitochondrial ATP production and cellular energy metabolism

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While our in vitro developability assessments were performed on a small cohort of target-specific, high-expression antibodies from each repertoire and requires validation in a larger, more unbiased subset to definitively attribute native pairing to gains in developability, the bioinformatic workflows employedparticularly our clustering-based clonal selection strategiessuggest that the selected antibodies should be representative in sequence and function of the larger target-specific diversity