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Species such as Lactobacillus , Bifidobacterium , Bacteroides , and Clostridium generate AhR agonists that can suppress the pro-inflammatory phenotype of glial cells [163, 165]

Similar to previous studies examining ACE2 from humans and other species 5,6,35,36 , elastase-activated RaTG13 and Khosta-2 spike proteins were shown to enter host cells via ACE2