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The excessive ROS production during reperfusion can lead to the opening of the mitochondrial permeability transition pore (mPTP), causing mitochondrial swelling, loss of membrane potential, and cell death [2]

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Handb Exp Pharmacol 196:369405 Holme JA, Hongslo JK, Bjrge C, Nelson SD (1991) Comparative cytotoxic effects of acetaminophen ( N -acetyl- p -aminophenol), a non-hepatotoxic regioisomer acetyl- m -aminophenol and their postulated reactive hydroquinone and quinone metabolites in monolayer cultures of mouse hepatocytes